2012 Unconventional roles of nonlipidated LC3 in ERAD tuning and coronavirus infection

4 Pages·2012·0.89 MB·English

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Date: 09 November 2015, At: 23:20

Autophagy

ISSN: 1554-8627 (Print) 1554-8635 (Online) Journal homepage: http://www.tandfonline.com/loi/kaup20

Unconventional roles of nonlipidated LC3 in ERAD
tuning and coronavirus infection

Riccardo Bernasconi, Julia Noack & Maurizio Molinari

To cite this article: Riccardo Bernasconi, Julia Noack & Maurizio Molinari (2012)
Unconventional roles of nonlipidated LC3 in ERAD tuning and coronavirus infection,
Autophagy, 8:10, 1534-1536, DOI: 10.4161/auto.21229

To link to this article: http://dx.doi.org/10.4161/auto.21229

Published online: 16 Aug 2012.

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©2012 Landes Bioscience. Do not distribute.

Autophagy 8:10, 1534–1536; October 2012; © 2012 Landes Bioscience

AutOphAgic punctum

1534
Autophagy
Volume 8 issue 10

Punctum to: Bernasconi R, Galli C, Noack J,
Bianchi S, de Haan CAM, Reggiori F, et al. Role
of the SEL1L:LC3-I complex as an ERAD tun-
ing receptor in the mammalian ER. Mol Cell
2012; 46:809–19; PMID:22633958; http://dx.doi.
org/10.1016/j.molcel.2012.04.017

Keywords: endoplasmic reticulum,
protein folding, protein quality control,
ERAD, ERAD tuning, autophagy,
EDEMosomes, ERAD tuning vesicles,
proteostasis, EDEM1, SEL1L, LC3-I,
coronaviruses

Submitted: 06/07/12

Revised: 06/21/12

Accepted: 06/22/12

http://dx.doi.org/10.4161/auto.21229

*Correspondence to: Maurizio Molinari;
Email: